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Conclusion:Digestion of rat spinal cord by 0.125% trypsin for 45 and 60 minutes could collect neural stem cells most effectively.
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结论:0.125%胰蛋白酶37℃振荡消化45、60min可以最有效的分离大鼠脊髓来源神经干细胞。 |
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Conclusion:Highly mixed DC/Th0 ration induced Th0 differentiation to Th1, low mixed DC/Th0 ration induced Th0 differentiation to Th2, DC regulating immunology response type were plastic which provided the basis for clinic GVHD therapy.
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低比例混合时 ,诱导Th0向Th2分化。 显示人外周血DC在调控T淋巴细胞免疫应答类型上具有可塑性 ,为临床移植物抗宿主病 (GVHD)免疫干预治疗奠定基础。 |
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Conclusion:This FQ-PCR provides a more efficient method for measles virus RNA detection.
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结论:FQ-PCR法为麻疹病毒核酸检测提供了一种可靠的方法。 |
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ConclusionThe expression of hMLH1and hMSH2 was correlated with differentiation.
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结论MLH1、hMSH2的表达只与分化程度相关,随分化程度的降低,其失表达越明显。 |
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Conclusion\ mGluR1,ER and IL 2 can coexist in same rat central neurons,therefore,providing morphological basis for the theory about immune neuro endocrine network at the cellular level for the first time.
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结论在大鼠中枢神经系统内mGluR1、ER和IL-2可共存于同一个神经细胞,从而首次在细胞水平为神经免疫内分泌网络学说提供了形态学依据。 |
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Conclusions 1.In Pca tissues the VEGF-C positive expression rate increases along with the decreased tumor differentiation degree, and prompts that the VEGF-C protein expression has concern with the Pca cell growth and differentiation degree.
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结论(1)Pca组织中VEGF-C阳性表达率,随肿瘤分化程度的降低而明显增高,提示VEGF-C蛋白表达与Pca细胞生长及分化程度有关。 |
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Conclusions Compound lactobacilli could improve the balance between inflammatory and anti-inflammation cytokines as well as the intestinal immune function in SAP rats.
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结论复合乳酸菌可改善SAP大鼠促炎和抗炎细胞因子的平衡状态和肠道的免疫功能; |
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Conclusions EBV might be a role in the pathogenesis of lymphoblastic NK/T cell lymphomas.
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结论 EBV在母细胞性NK/T细胞淋巴瘤的发生中可能具有一定作用。 |
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Conclusions Genes related with Galpha13 signaling pathway differently expressed in ED14.5 Cx43 knockout OFT tissue may be involved in the pathogenesis of pulmonary outflow obstruction.
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结论利用基因芯片技术初步筛选出与Cx43KO鼠胚心脏近端流出道发育有关的多个基因,其中Galpha13信号通路上的相关基因可能与Cx43KO小鼠流出道梗阻的发生有关。 |
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Conclusions Regulatory T cells possibly plays an important role by cytokines such as TGF-β, IL-10, IL-12 in the pathogenesis of SLE.
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结论调节性T细胞可能通过细胞因子TGF-β、IL-10、IL-12参与SLE的发病机制,但具体机制有待于进一步研究。 |
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Conclusions TNF-α,MCP-1 are cytokines which were activated in sepsis early,and their release is related with NF-κB activity.
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结论TNF-α、MCP-1是脓毒症早期激活的细胞因子,它们的释放与NF-κB的活性密切相关; |