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CONCLUSION The method is sensitive and accurate for the determination of busulfan concentration in human plasma. It is suitable for the pharmacokinetics study of busulfan.
中文意思:
结论:该方法准确、灵敏,适用于白消安血浆浓度测定及其药动学研究。


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COMPARISON OF c-myc GENE EXPRESSION IN HL-60 LEUKEMIA CELLS BY RA AND A LOW MOLECULAR TUMOR SUPPRESSOR OF HUMAN FETAL LIVER ORIGIN 胎肝低分子抑瘤物和维生素A酸对HL-60细胞中c-myc癌基因表达的影响
COMPARISONS ON THE MOLECULAR CHARACTERISTICS OF VIBRIO CHOLERAE O139 霍乱弧菌O139国内外菌株分子生物学特征比较
COMPLEX SYSTEMS MODELING VIA NEURO FUZZY METHOD 基于神经模糊方法的复杂系统建模
CONCLUSION Pharmacokinetics of 188 Re HAb18 F(ab) 2 conforms to a l compartment model. 结论  1 88Re- HAb18F(ab ) 2 在荷肝癌裸鼠体内药代动力学符合线性一室模型
CONCLUSION The Hill coefficient is considered not any physiological meanings in the mathematics model usually used in fitting pharmacodynamics S dose-effect curve. 结论常用的药效学S型剂量效应曲线拟合的数学模型中,Hill系数无生理学含义。
CONCLUSION The method is sensitive and accurate for the determination of busulfan concentration in human plasma. It is suitable for the pharmacokinetics study of busulfan. 结论:该方法准确、灵敏,适用于白消安血浆浓度测定及其药动学研究。
CONCLUSION This method was simple,quick,and accurate and can be used for studing the pharmacokinetics of tegafur and therapeutic drug monitoring. 结论本方法简单、快速、准确,适用于临床药动学研究及血药浓度监测。
CONCLUSION Yizhikang could decrease the level of serum TC, TG, LDL-C, VLDL-C in rabbits with experimental endogenous or exogenous hyperlipidemia and regulate the level of blood lipids. 结论益脂康可显著降低内源性和外源性高家兔高血脂模型TC、TG、LDL-C、VLDL-C,调节血脂。
CONCLUSION: BMSCs can differentiate into hepatocytes under the induction of HE in vitro, and the transplantation of BMSCs can be helpful in the repair of injured liver in mice. 结论:BMSCs在体外可由HE诱导分化成肝细胞,在体内移植后可促进小鼠受损肝脏的修复.
CONCLUSION: Expression of cytokeratin 19 is observed during the differentiation of pancreas stem cells and cytokeratin 19 is a sign of pancreas stem cells. 结论:胰腺干细胞在分化过程中有细胞角蛋白19表达,细胞角蛋白19是胰腺干细胞的标志物。
CONCLUSION: Extracellular Ca2+ influx into fibroblasts increased collagen production, However, the DNA synthesis was suppressed when the cytosolic Ca2+ was too high or too low. 结论:细胞外液Ca~(2+)内流促进胶原合成,细胞内Ca~(2+)浓度过高或过低均抑制脱氧核糖核酸合成。
 
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