|
Conclusions The tendency of lowering of immunoglobulin,lymphocyte count,NK cell count,and CD4/CD8 ratio indicate that there is a rapid deterioration of both cellular and humoral immunity after irradiation. |
中文意思: 结论大剂量照射后免疫球蛋白、淋巴细胞数、NK细胞数、CD4/CD8比值等总体呈下降趋势提示,照射后细胞及体液免疫功能均快速受损。 |
以下为相关句子列表(点击查看,可以看对应的解释):
|
|
Conclusions EBV might be a role in the pathogenesis of lymphoblastic NK/T cell lymphomas.
|
|
|
结论 EBV在母细胞性NK/T细胞淋巴瘤的发生中可能具有一定作用。 |
|
Conclusions Genes related with Galpha13 signaling pathway differently expressed in ED14.5 Cx43 knockout OFT tissue may be involved in the pathogenesis of pulmonary outflow obstruction.
|
|
|
结论利用基因芯片技术初步筛选出与Cx43KO鼠胚心脏近端流出道发育有关的多个基因,其中Galpha13信号通路上的相关基因可能与Cx43KO小鼠流出道梗阻的发生有关。 |
|
Conclusions Regulatory T cells possibly plays an important role by cytokines such as TGF-β, IL-10, IL-12 in the pathogenesis of SLE.
|
|
|
结论调节性T细胞可能通过细胞因子TGF-β、IL-10、IL-12参与SLE的发病机制,但具体机制有待于进一步研究。 |
|
Conclusions TNF-α,MCP-1 are cytokines which were activated in sepsis early,and their release is related with NF-κB activity.
|
|
|
结论TNF-α、MCP-1是脓毒症早期激活的细胞因子,它们的释放与NF-κB的活性密切相关; |
|
Conclusions TNF-αand IL-6 were the major inflammatory cytokines released in the early phase of MODS,and played an important role in the delayed-development of it.
|
|
|
结论TNF-α、IL-6是MODS早期释放的致炎细胞因子,在MODS的发生、发展中起非常重要的作用。 |
|
Conclusions The tendency of lowering of immunoglobulin,lymphocyte count,NK cell count,and CD4/CD8 ratio indicate that there is a rapid deterioration of both cellular and humoral immunity after irradiation.
|
|
|
结论大剂量照射后免疫球蛋白、淋巴细胞数、NK细胞数、CD4/CD8比值等总体呈下降趋势提示,照射后细胞及体液免疫功能均快速受损。 |
|
Conclusions There might exist a gut-liver-lung axis in the kinetics of expression and release of major pro-inflammatory cytokines after hemorrhagic shock.
|
|
|
肺脏间质水肿、炎症细胞浸润。 结论:失血性休克时细胞因子的释放顺序是肠道、肝脏和肺脏,推测存在”肠-肝-肺”细胞因子释放轴的可能,有待进一步确定。 |
|
Conclusions the positive rates of enterovirus RNA in PM/DM patients are high, which is related to the elevation of CPK and muscular pain.
|
|
|
结论 肠道病毒核糖核酸在 PM/DM患者中检出率高; |
|
Conclusions: Cytokines (TNF-α, IL-1β, IFN-γ) enhance TLR4, MD-2 expression and promote the reaction with LPS, leading to overactivity of IEC with commensal bacteria, and accordingly initiate or perpetuate the intestinal inflammation.
|
|
|
结论:细胞因子(TNF-α,IL-1β,IFN-γ)可增加肠上皮细胞TLR4和MD-2的表达,促进其对LPS的反应,引起肠上皮细胞对常驻菌的过度反应,从而启动或加重肠道炎症。 |
|
Condusion Recombined rAAV2/eGFP and rAAV2/NGF virus can be transfected into neural stem cells which could express GFP,NGF steadly and may play a important role in therapy of nervous system diseases.
|
|
|
结论 重组rAAV2 /eGFP和rAAV2 /NGF病毒可转染神经干细胞,神经干细胞可稳定表达GFP和NGF ,可用于基因治疗神经系统疾病。 |
|
Consideration about Journal of Immunology's Entering International Retrieval System
|
|
|
关于《免疫学杂志》进入国际检索系统的思考 |
|
|
|